Chronic Fatigue, ME and Fibromyalgia by Alison Adams

Author:Alison Adams
Language: eng
Format: epub
Tags: Chronic Fatigue, ME and Fibromyalgia: The Natural Recovery Plan
ISBN: 9781780284576
Publisher: Duncan Baird Publishing
Published: 2009-09-25T04:00:00+00:00

The Effects of Mercury Toxicity

The first system affected by mercury toxicity is the immune system where the number of natural killer cells are reduced and thus become unable to police tumours and virus-infected cells. Various yeasts and bacteria such as Candida albicans and Escherichia coli are known to have the protective capacity to convert inorganic mercury into methyl mercury. When the immune neutrophils engulf these mercury-laden organisms they are disabled, losing both their motility and effectiveness. In this manner mercury toxicity can promote the overgrowth of yeasts which can ultimately become systemic, invading every organ and system. Intervention using antibiotics may unwittingly inflame this scenario as they obliterate the friendly bacteria controlling yeast growth – although this may not be apparent until months or years later. The affected neutrophils inevitably die as a result of consuming the mercury toxic yeasts and are dismantled in the liver where the mercury may effectively concentrate. Mercury toxicity also increases the ratios of T helper cells (which initiate an immune response) to T suppressor cells (which terminate an immune response) and this may lead to the development of allergies.

Significantly, mercury combines with the ‘bar code’ (the major histocompatibility complex) on the cell membrane that identifies cells as being ‘self’ and in so doing changes the bar code to ‘non-self’. This results in the immune system attacking body cells as in the autoimmune diseases such as multiple sclerosis, Parkinson’s disease, diabetes and Hashimoto’s thyroiditis.

Methyl mercury also prevents cell division and generation and interferes with the formation of the structural tubulin in the microtubules of the cell and neurones and the formation of the insulating myelin sheaths around nerves. These factors, when combined with the lipophilic (fat-loving) and neurophilic (nerve-loving) nature of methyl mercury, explain the devastating and long-lasting effects both in the developing foetus and child (where tissues are growing rapidly) and in the tremors and psychological and behavioural effects in the adult. Mercury profoundly impacts the management of fats in the body too, altering steroid and cholesterol regulation and destroying and oxidizing both essential fatty acids and the phospholipids of the cell membrane. Mercury toxicity may promote elevated blood cholesterol levels (which may be protective) and ultimately cardiovascular problems. Therefore, seeking to lower cholesterol levels is treating a symptom rather than the silent and unseen cause.

Mercury also affects the mitochondria which, if deprived of adequate oxygen, switch to anaerobic energy production with the associated build-up of lactic acid which causes the muscle stiffness and aching that are familiar to most FRS sufferers. The mitochondria become unable to recycle the spent ADP back to ATP efficiently as mercury disables a key enzyme and this results in further breakdown within the cytosol into adenosine mono-phosphate (AMP) which is then excreted in the urine. It then takes up to four days to synthesize ‘new’ ATP from glucose and this may account for the post-exertional malaise.

Even the proponents of the use of mercury in dentistry admit that elevated amounts remain detectable in the blood for up to three months after an amalgam filling is either removed or placed.

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